Study of Right Ventricular Pacing Threshold at National Teaching Hospital of Cotonou in Benin
American Journal of Internal Medicine
Volume 6, Issue 3, May 2018, Pages: 47-51
Received: May 2, 2018;
Accepted: May 21, 2018;
Published: Jun. 14, 2018
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Arnaud Sonou, Department of Cardiology, Departmental Teaching Hospital of Ouémé-Plateau, University of Abomey-Calavi, Porto-Novo, Benin
Tchaa Tchérou, Department of Cardiology, University Teaching Hospital of “Campus”, Lomé, Togo
Philippe Mahouna Adjagba, Department of Cardiology, Teaching Hospital of Cotonou, University of Abomey-Calavi, Cotonou, Benin
Murielle Hounkponou, Department of Cardiology, Teaching Hospital of Cotonou, University of Abomey-Calavi, Cotonou, Benin
Léopold Codjo, Department of Cardiology, Departmental Teaching Hospital of Borgou-Alibori, University of Parakou, Parakou, Benin
Salimatou Assani, Department of Cardiology, Teaching Hospital of Cotonou, University of Abomey-Calavi, Cotonou, Benin
Wilfried Gandji, Department of Cardiology, Teaching Hospital of Cotonou, University of Abomey-Calavi, Cotonou, Benin
Yessoufou Tchabi, Department of Cardiology, Teaching Hospital of Cotonou, University of Abomey-Calavi, Cotonou, Benin
Martin Houénassi, Department of Cardiology, Teaching Hospital of Cotonou, University of Abomey-Calavi, Cotonou, Benin
The main purpose was to study the right ventricular pacing threshold of patients who benefit from cardiac pacemaker’s implantation at the National Teaching Hospital of Cotonou in Benin. This was a retrospective study from January 2007 to September 2013. The right ventricular pacing threshold measured during successive checks has been studied. A threshold greater than 1 volt was considered high. The conventional causes of threshold’s rising were sought. 35 files were examined. The average threshold of our patients amounted to 1.24 ± 1.17 volts at the first control and remained high until the end of follow-up. The prevalence of threshold elevation was 17% (6/35). The causes of threshold elevation were: late displacement of the lead (1 case), faulty lead (1 case), and suspected lead’s micro dislodgment (1 case). There was no formal etiology found for 3 cases. Half of threshold elevation cases involved a problem of pacing lead. Similar findings have been reported by previous studies. This study has confirmed that threshold elevation mainly involved pacemaker lead. The premature need to change the pacemaker caused by this elevation has considerable consequences in a Beninese local context marked the lack of governmental facilities.
Philippe Mahouna Adjagba,
Study of Right Ventricular Pacing Threshold at National Teaching Hospital of Cotonou in Benin, American Journal of Internal Medicine.
Vol. 6, No. 3,
2018, pp. 47-51.
Maisel WH. Pacemaker and ICD generator reliability: meta-analysis of device registries. JAMA 2006; 295: 1929-34.
Kane A. et coll. Problematique de la stimulation cardiaque definitive en Afrique subsaharienne: étude multicentrique STIMAFRIQUE. Cardiologie tropicale 2016; 23 (143): 20.
Adoubi KA, Kendja KF, Tano M, Koffi F, Ndjessan JJ, Meneas C et coll. Activities report of Abidjan Cardiology Institute pacing Unit from 2006 to 2012. Cardiovascular Journal of Africa 2013; 24 (5).
Jouven X. La stimulation cardiaque en Afrique de l’Ouest. Annales de Cardiologie et d’Angéiologie 2003; 52 (4): 204.
Bouraoui H, Trimech B, Chouchene S, Mahdhaoui A, Hajri SE, Jeridi G et coll. La stimulation cardiaque permanente: à propos de 234 patients. La Tunisie Medicale 2011; 89 (07): 604–609.
Sdiri W, Marouf A, Mbarek D, Ben Slima H, Mokaddem A, Ben Ameur Y et coll. Résultats de la stimulation cardiaque définitive: à propos de 188 malades. La tunisie Médicale 2013; 91 (06): 396–401.
Udo EO, Van Hemel NM, Peter N, Zuithoff A, Nijboer H, Taks W and al. Incidence and predictors of shortand long-term complications in pacemaker therapy: The FollowPace study. Heart Rhythm. 2012; 9(5):728-35.
Hai-Bo Y, Yan-Chun L, Guo-Qing X. The Comparison Between the Active-Fixation Leads and Passive-Fixation Leads in Right Ventricular Apical Pacing. Heart 2013; 99:A8-A9. doi:10.1136/heartjnl-2013-303992.027.
Liu L, Tang J. A long-term, prospective, cohort study on the performance of right ventricular pacing leads: comparison of active-fixation with passive-fixation leads. Scientific Reports 2015, 5: 7662. doi: 10.1038/srep07662.
Aggarwal RK, Connelly DT, Ray SG, Ball J, Charles RG. Early complications of permanent pacemaker implantation: no difference between dual and single chamber systems. Br HeartJ1 1995; 73:571-575.
Peter O. Adeoye, Kelechi E. Okonta, Mudasiru A. Salami, Victor O. Adegboye. Experience with permanent pacemaker insertion at the University College Hospital, Ibadan, Nigeria. Nigerian Journal of Cardiology 2013; 10 (1): 3-5.
Barold S, Falkoff M, Ling S, Robert A. Hyperkalemia-Induced Failure of Atrial Capture during Dual-Chamber Cardiac Pacing. J Am Coll Cardiol 1987; 10:467-9.
Takasugi N, Kubota T, Kawamura I, Takasugi M, Kanamori H. Sudden reversible pacemaker failure in a patient with cardiac sarcoidosis: an unfortunate case of ventricular septal pacing. Doi: 10.1093/eupace 2012/eur435.
Sperzel J, Burri H, Gras D, Tjong FVY, Knops ER, Hindricks G and al. State of the art of leadless pacing. Europace 2015; 17:1508–1513.
Paul A. Levine. Management of the Patient with an Acute Massive Rise in the Capture Threshold. Indian Pacing and Electrophysiology Journal 2001, 1(1): 35-37.
Nagatomo Y, Ogawa T, Kumagae H, Koiwaya Y, Tanaka K. Pacing failure due to markedly increased stimulation threshold two years after implantation: successful management with oral prednisolone. PACE 1989; 12: 1034-1037.